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Additional features: hyperlipidemia; insulin resistance; hyperinsulinemia; hyperglycemia; low levels of high density lipoprotein; and increased risk of diabetes mellitus and atherosclerosis. Mechanisms for development are not completely understood. HIV-1 protease inhibitor drugs and nucleoside reverse transcriptase inhibitors are implicated as follows: decreased production of retinoic acid and triglyceride uptake; inhibition of mitochondrial DNA (mtDNA) polymerase gamma; inhibition of lipid metabolism; and prevention of development of adipocytes. Evidence suggests decreased insulin sensitivity, beta-cell dysfunction, and down-regulated estrogen receptor expression in adipose tissue. In absence of HAART, HIV-1 may itself cause dyslipidemia and lipodystrophy by various mechanisms. Resting energy expenditure and lipid oxidation are higher in HAART-treated HIV-positive patients with vs. without lipodystrophy."],"label":[],"synonym":[],"obsolete":false,"prefixIRI":null,"subClassOf":[],"xref":null,"inScheme":[],"created":null,"modified":null,"memberOf":[],"altLabelXl":[],"hiddenLabelXl":[],"parents":[],"prefLabelXl":[],"isInActiveScheme":[null],"isInActiveCollection":[],"properties":{"http://purl.bioontology.org/ontology/MEDDRA/SMQ_STATUS":["A"],"http://www.w3.org/1999/02/22-rdf-syntax-ns#type":["http://www.w3.org/2002/07/owl#Class"],"http://bioportal.bioontology.org/ontologies/umls/tui":["T185"],"http://www.w3.org/2004/02/skos/core#notation":["20000177"],"http://purl.bioontology.org/ontology/MEDDRA/SMQ_LEVEL":["1"],"http://bioportal.bioontology.org/ontologies/umls/hasSTY":["http://purl.bioontology.org/ontology/STY/T185"],"http://purl.bioontology.org/ontology/MEDDRA/SMQ_ALGO":["N"],"http://bioportal.bioontology.org/ontologies/umls/cui":["C2721746"],"http://www.w3.org/2004/02/skos/core#prefLabel":["Lipodystrophy (SMQ)"],"http://purl.bioontology.org/ontology/MEDDRA/SMQ_SOURCE":["Robles, DT. Lipodystrophy, HIV. eMedicine, 24 June 2008 (http://www.emedicine.com/derm/topic877.htm); WHO/Forum for Collaborative HIV Research Joint Meeting: ARV Drugs Adverse Events, Case Definition, Grading, Laboratory Diagnosis and Treatment Monitoring, Background Document (Draft), 28 - 29 February 2008"],"http://www.w3.org/2004/02/skos/core#definition":["HIV associated lipodystrophy: abnormal central fat accumulation (lipohypertrophy) and localized loss of fat tissue (lipoatrophy). Some patients have only lipohypertrophy or only lipoatrophy; others have a mixed clinical presentation. Lipohypertrophy and lipoatrophy are distinct entities with different risk factors and underlying metabolic processes. Lipohypertrophy: enlarged dorsocervical fat pad; circumferential expansion of neck; breast enlargement (including gynecomastia); abdominal visceral fat accumulation and other new fat accumulations that are circumscribed (e.g., lipomas) or general. Lipoatrophy: peripheral fat wasting; loss of subcutaneous tissue in face, arms, legs, buttocks; involvement of face is most common. Additional features: hyperlipidemia; insulin resistance; hyperinsulinemia; hyperglycemia; low levels of high density lipoprotein; and increased risk of diabetes mellitus and atherosclerosis. Mechanisms for development are not completely understood. HIV-1 protease inhibitor drugs and nucleoside reverse transcriptase inhibitors are implicated as follows: decreased production of retinoic acid and triglyceride uptake; inhibition of mitochondrial DNA (mtDNA) polymerase gamma; inhibition of lipid metabolism; and prevention of development of adipocytes. Evidence suggests decreased insulin sensitivity, beta-cell dysfunction, and down-regulated estrogen receptor expression in adipose tissue. In absence of HAART, HIV-1 may itself cause dyslipidemia and lipodystrophy by various mechanisms. 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